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  Indian J Med Microbiol
 

Figure 1: The mechanism of hyperglycemia after stroke. Increased hepatic gluconeogenesis and decreased insulin sensitivity are the causes of poststroke hyperglycemia. The application of β-adrenergic antagonist can reduce insulin resistance. By enhancing the effect of STAT3: signal transducers and activator of transcription-3, IL-13 inhibits the gene transcription for gluconeogenesis and downregulates gluconeogenesis. See the main text for more specific regulatory pathways. IL-13: Interleukin-13

Figure 1: The mechanism of hyperglycemia after stroke. Increased hepatic gluconeogenesis and decreased insulin sensitivity are the causes of poststroke hyperglycemia. The application of β-adrenergic antagonist can reduce insulin resistance. By enhancing the effect of STAT3: signal transducers and activator of transcription-3, IL-13 inhibits the gene transcription for gluconeogenesis and downregulates gluconeogenesis. See the main text for more specific regulatory pathways. IL-13: Interleukin-13